Search results for "Sphingosine kinase 1"

showing 4 items of 4 documents

Cardioprotection by gene therapy

2015

Ischemic heart disease remains the leading cause of death worldwide. Ischemic pre-, post-, and remote conditionings trigger endogenous cardioprotection that renders the heart resistant to ischemic-reperfusion injury (IRI). Mimicking endogenous cardioprotection by modulating genes involved in cardioprotective signal transduction provides an opportunity to reproduce endogenous cardioprotection with better possibilities of translation into the clinical setting. Genes and signaling pathways by which conditioning maneuvers exert their effects on the heart are partially understood. This is due to the targeted approach that allowed identifying one or a few genes associated with IRI and cardioprote…

Cardioprotectionmedicine.medical_specialtybiologybusiness.industryGene targetingSphingosine kinase 1Heat shock proteinInternal medicineGene expressionmedicinebiology.proteinCardiologyHepatocyte growth factorSignal transductionCardiology and Cardiovascular MedicinebusinessTranscription factormedicine.drugInternational Journal of Cardiology
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An active form of sphingosine kinase-1 is released in the extracellular medium as component of membrane vesicles shed by two human tumor cell line.

2010

Expression of sphingosine kinase-1 (SphK-1) correlates with a poor survival rate of tumor patients. This effect is probably due to the ability of SphK-1 to be released into the extracellular medium where it catalyzes the biosynthesis of sphingosine-1-phosphate (S1P), a signaling molecule endowed with profound proangiogenic effects. SphK-1 is a leaderless protein which is secreted by an unconventional mechanism. In this paper, we will show that in human hepatocarcinoma Sk-Hep1 cells, extracellular signaling is followed by targeting the enzyme to the cell surface and parallels targeting of FGF-2 to the budding vesicles. We will also show that SphK-1 is present in a catalitycally active form i…

Article SubjectNeutral CeramidasebiologySphingosineVesicleCellmembrane vesicleslcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensSphK vesicleslcsh:RC254-282Cell biologychemistry.chemical_compoundmedicine.anatomical_structureOncologySphingosine kinase 1chemistryBiosynthesisCell cultureSettore BIO/10 - Biochimicabiology.proteinExtracellularmedicinesphingosine kinase; ceramidase; tumoe cells. microvesiclesResearch Article
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The sphingosine kinase 1 activator, K6PC-5, attenuates Ebola virus infection

2021

Summary Ebola virus (EBOV) is responsible for outbreaks with case fatality rates of up to 90% and for an epidemic in West Africa with more than ten thousand deaths. EBOV glycoprotein (EBOV-GP) is the only viral surface protein and is responsible for viral entry into cells. Here, by employing pseudotyped EBOV-GP viral particles, we uncover a critical role for sphingolipids in inhibiting viral entry. Sphingosine kinase 1 (SphK1) catalyzes the phosphorylation of sphingosine to sphingosine 1-phosphate (S1P). The administration of the SphK1 activator, K6PC-5, or S1P, or the overexpression of SphK1 consistently exhibited striking inhibitory effects in EBOV-GP-driven entry in diverse cell lines. F…

0301 basic medicineScienceviruses02 engineering and technologymedicine.disease_causeArticle03 medical and health scienceschemistry.chemical_compoundViral entryVirologymedicinechemistry.chemical_classificationMultidisciplinaryEbola virusSphingosinebiologyActivator (genetics)QMolecular MicrobiologyCell Biology021001 nanoscience & nanotechnologyVirologySphingolipid030104 developmental biologychemistrySphingosine kinase 1Cell culturebiology.protein0210 nano-technologyGlycoproteiniScience
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The Sphingosine Kinase-1 Activator, K6PC-5, Attenuates the Ebola Virus Infection and the Virus Induced Cell Death

2020

Ebola virus (EBOV) is responsible for outbreaks with case-fatality rates of up to 90% and for an epidemic in West Africa with more than ten thousand deaths. EBOV glycoprotein (EBOV-GP) is the only viral surface protein and is responsible for viral entry into cells. It has been suggested to play a role in the cytopathic effects induced by the virus. Here we uncover a critical role for sphingolipids in inhibiting viral entry and virus-mediated cytotoxicity. Sphingosine kinase 1 (SphK1) catalyzes the phosphorylation of sphingosine to sphingosine-1-phosphate (S1P).  The administration of the SphK1 activator, K6PC-5, or S1P, or the overexpression of SphK1 consistently exhibited striking inhibito…

Programmed cell deathEbola virusSphingosineActivator (genetics)Biologymedicine.disease_causeVirologySphingolipidViruschemistry.chemical_compoundSphingosine kinase 1chemistryViral entrybiology.proteinmedicineSSRN Electronic Journal
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